Apr 24, 2015 | Ailments, Feet, Hoof, Hoof Care, Lameness, Laminitis, Leg issues, Shoeing
Part 2: Everything You Need to Know About Your Horse’s Feet
So they made me do a little work between blogs but here is Part 2 of my exclusive report from the All You Need to Know About Your Horse’s Foot Seminar.
When you hear ‘break-over’ you should think of thunderclouds parting and the sun coming out, angels singing, a drink of water when you are really thirsty. It’s that important. Horses have multiple break-overs, but we are going to concentrate on the one at the front of the foot for now. A break-over is exactly what it sounds like. It’s the point at which the hoof comes off the ground during movement. The moment of break over is the hardest on the hoof structures. Every bit of that force is trying to tear the foot apart.
A good trim, according to the guidelines I talked about in Part I, will set up a good break-over. This point should be about ½” to ¾” in front of the tip of the coffin bone. Wait a minute… how is my farrier supposed to know where the tip of the coffin bone is located?? I don’t think they come equipped with x-ray vision! Most of the time your farrier will set the break-over at a point about ½” to ¾” in front of the tip of the frog. Sometimes the foot doesn’t seem to be doing what your farrier expects. X-rays of the foot will help your farrier see what the bones are doing inside the hoof. X-rays let us see if there is arthritis, injuries, or laminitis going on which may require special shoeing.
Visual Aids!
Let’s look at some feet and see if you can spot the problem. First up:
This guy definitely doesn’t have his heels back to the widest part of his frog and that’s with the shoe on. And if you look at the branches of the shoe you will see they aren’t even. The shoe is twisted on the foot. His break-over is also too far forward.
Next up:
A line drawn down the center of this foot definitely won’t give you half inside and half outside.
This shoe…..Unless half this shoe was on one foot and half was on another foot it doesn’t make sense.
Now for the fun stuff. What happens when the foot has been trimmed and we can’t get the alignment the way we want it? We put a shoe on it!! So in answer to the barefoot question. Your horse can go barefoot if two things can happen: the foot can be balanced with a trim, and the work the horse is doing doesn’t unbalance the foot faster than it can grow to compensate.
This shoe is an extreme example of break-over manipulation. It starts with a regular shoe. Bar stock is then welded to the inside. Then the shoe is nailed to the foot. The end result is a shoe that allows this horse to break-over anywhere he wants.
This horse is an example of the opposite end of the spectrum. This foot has been allowed to grow long to bring the break-over forward but notice the principles have still been applied. Now this foot is extra long since this horse is due to be shod but notice the foot is balanced and well supported. We can manipulate feet to make gaits we find appealing but it must be done correctly or we jeopardize the horse.
Ever get tired of your young horse pulling shoes? Or have a horse with really sore heels? This shoe, affectionately called the flip flop, fully supports the foot but is very forgiving of the hind foot grab. Most short coupled young horses go through a shoe pulling phase until they learn to wear shoes. It’s normal and should not be blamed on your farrier. Sore-heeled horses need some support since they hurt too bad to not have a shoe underneath them, but metal shoes can be too hard on them. The heels on these shoes offer them soft support.
This shoe is an example of the compromises we have to make sometimes. This horse has a torn deep digital flexor tendon. This injury will heal best if we can take some of the tension off the tendon. A shoe like this helps reduce that tension but it has moved that pressure to the heels. Notice how the heel is curving under. This shoe can’t be used long term without causing significant hoof problems.
A quick note on some common Florida problems. This beautiful abscess is secondary to all the very wet weather we have been having. Using durasole, thrush buster, keratex hoof hardener or something similar on the foot will help it handle all that excess moisture. Sometimes no matter what we use on the foot they just stay way too wet. In that situation shoes may be the answer if only temporarily.
Along with abscesses, thrush is a common problem. The cattle mastitis ointment Today applied to the affected areas daily for a week and then every few days for three weeks will help clear it up. We have also recently started using a product called Keramend on some really really really bad thrush horses and have been very pleased with the results.
Our horses rely on a good foundation to stay happy and comfortable. I know I learned a lot about feet at this seminar, but Dr. Lacher and the entire Springhill Equine crew are happy to talk feet anytime! This is Tony saying may your food bowl be full and your litter box clean!!
Apr 13, 2015 | Feet, Hoof, Hoof Care, Lameness, Laminitis, Leg issues, Shoeing
This is Tony reporting from the Hoof Seminar on April 11, 2015. I am sure this will be a multi-part blog since I doubt I will be able to get all the information typed up without hitting the food bowl at least once so stay tuned if I stop mid-sentence. I will be back after refreshments….
Let’s start with the very true saying: “No Hoof No Horse.” Much like “You can lead a horse to water but you can’t make them drink” this saying sticks around because it is so true. Our horse’s take off, land, turn, and stand on their feet. And it’s a pretty tiny foot on the end of a long stick so little changes make huge differences!
A bit of anatomy will help us all get our bearings for some good discussion later. The lower leg is made up of the cannon bone, sesamoids, long pastern, short pastern, navicular bone, and coffin bone. The most important thing to remember here is that we are manipulating the hoof since that’s the part we can change but it has to align with all those bone and tendons and ligaments above.
Moving on to alignment. Tires on your car have to be aligned a certain way so they wear evenly. Hooves aren’t much different. By following certain guidelines the hoof will fly through the air, land, and take off again in perfect alignment with the rest of the limb above, the other three legs, and the big body it’s moving along. Think about it: a horse trotting creates 1,500 pounds of pressure and that’s at the trot! It’s really important to get alignment right.
OK starting with the view from side…….A line drawn down the front of the hoof should lay against the entire hoof wall and front of pastern. A line drawn down from the center of the fetlock joint should lay against the heel bulbs. In a perfect world the fetlock and hoof wall line would intersect at the middle of the fetlock joint but that is where the word guideline comes in.
Now pick up your horse’s foot. When it is freshly trimmed the heels should be at the widest point of the frog. This is really, really, really important. Many of us where taught that heel should never be taken off. Like many things we heard in our youth, things changed. The heels must be at the widest point of the frog. Next draw a line across the widest part of the foot and a line at the toe. Half the foot should be in front of the widest point and half behind.
Draw a line down the center of the foot long ways and half the foot should be to the outside, half to the inside.
Set the foot back down and look at it from the front. A line drawn down the center of the hoof should divide the hoof in half with 50% to the inside, 50% to the outside.
Hold your horse’s hoof up and let it hang from your hand. Draw a line down the center of the heels and again this should divide the hoof in half. This line should also be perpendicular to a line drawn across the heels where they will meet the ground. I hope you are noticing a pattern here. The foot should be symmetrical!
Notice none of this involved touching the sole with a hoof knife! By allowing our horses to form all the sole they want we help them keep good cushion beneath their coffin bone! Horses will naturally wear off any sole they don’t need so we don’t have to do that for them.
All this work has set us up for a discussion about the perfect break over, which is our real goal but you will have to wait until next week since I hear the full food bowl and clean litter box calling. Stay tuned. I will finish writing soon!
Apr 7, 2015 | Uncategorized
We cherish our older horses. In some cases we have a long history and lots of wonderful memories with them. In others, we value their kind and quiet personalities with our kids and (not-so-horsey) spouses. However they came in to our lives, the Seniors often show us the best qualities of horses and as ambassadors for the equine world they deserve the best in care. Caring for senior horses is easier than ever with modern feeds, health monitoring, and medications.
Let’s start with health monitoring. Providing regular dental care to your young horse means good teeth for many years to come. Dr. Lacher posted a picture of her 30 year old horse to Facebook demonstrating his excellent teeth. Just last week she saw another 25 year old horse who had received great dental care her whole life and as a result had perfect teeth. A good set of chompers means these two horses can eat normal feed and roughage keeping feeding costs down, maintain a happier gut, and experience a higher quality of life.
If your older horse didn’t have the luck to have a fantastic owner is his younger years don’t despair. Senior feeds and leafy hays keep these guys in on the action for a long time. These horses do experience more dental problems such as loose teeth and tooth root infections. A call to Springhill Equine at the slightest hint of dental pain allows us to manage most of these problems quickly and easily.
After dental disease, Equine Metabolic Syndrome (EMS) and Cushings disease are the next most common issues we face with our older horses. EMS and Cushings go together like peanuts and peanut butter most of the time. Wait isn’t it peanut butter and jelly? Yes but in this case EMS affects younger horses (as young as 3-4 years old) and over time many of those horses develop Cushings. So just like you start with peanuts to get to peanut butter, we often see EMS first and then see Cushings.
There are ways to identify EMS horses using blood tests but often we simply evaluate your horse’s body condition score (BCS). Horses who are a BCS of 7 or higher on a scale of 9 are almost always afflicted with EMS. This disease is similar to Type II Diabetes in humans and is managed with similar diet and exercise life changes. EMS leads to Cushings if not well managed because these horses are in a Pro-Inflammatory State. That means the immune system is always looking for places to take out its aggression and it doesn’t always find the right place to do that. If we are unsure if a horse has EMS we have a very simple blood test which can be performed: you, the owner, give about 100cc of Karo syrup and then about 90-120 minutes later we come out, draw blood, and send it off for insulin levels.
Cushings disease is recognized commonly in horses, dogs, and humans. Of course, horses have to be special by developing the disease in a different area of the pituitary gland than humans or dogs. Humans and dogs have a problem with the back of the pituitary gland or the adrenal glands, while horses develop their problem in the middle of the pituitary gland. What’s the big deal you ask? 5,000 different possible hormones is the big deal. While humans and dogs primarily release cortisol (the nasty stress hormone), your horse might release any combination of the 5,000 different hormones controlled by the middle of the pituitary gland. These hormones are primarily responsible for maintaining status quo in the body. They let the body know if it’s hot or cold, hungry or thirsty, should we grow feet or stop, etc. This means we can’t look at any given horse and say “Yep, You’ve got Cushings.” Luckily we have two great blood tests that give us pretty good answers. ACTH levels are a simple blood draw and if elevated we can reliably say “This horse has Cushings.” Because of that whole 5,000 hormones thing sometimes we have a strong suspicion that a horse has Cushings but ACTH levels come back normal. In that case we go a second test know as a TRH stimulation. This test involves a shot of TRH, waiting 15 minutes, and then another quick and easy blood draw to test ACTH levels. Horses who have Cushings disease are managed with a combination of medications depending on ACTH levels and symptoms.
Last but not least: The Dreaded L Word. Laminitis has the power to strike fear in to any horse owner’s heart. Watching our horses hobble around is almost as painful for us as it is for them. Turns out most laminitis is secondary to EMS and Cushings so good monitoring for these two conditions will dramatically reduce your horse’s chances of developing laminitis. Here at Springhill Equine we also strongly recommend radio-graphs (x-rays) of your senior horse’s feet every year. Many EMS horses suffer from very low levels of laminitis which cause slowly progressive changes in the feet. Often they don’t show us signs until they have progressed quite far. By taking radio-graphs yearly we can identify subtle cases early and form a plan with your farrier.
Our Senior horses have worked hard for us, in return let’s give them a great retirement! Springhill Equine’s Senior Wellness Program has been designed to evaluate all the common problems we talked about in this newsletter. If you would like more information or have questions about your Senior horse contact any of the Springhill Team.
Jan 16, 2015 | Ailments, Breeding
The 5 Panel Genetic Test for AQHA registration
The AQHA offers its members a genetic test for a panel of 5 different genetic diseases affecting Quarter Horses. The panel tests for Hyperkalemic Periodic Paralysis (HYPP), Polysaccharide Storage Myopathy type 1 (PSSM 1), Malignant Hyperthermia (MH), Hereditary Epidermal Regional Dermal Asthenia (HERDA), and Glycogen Branching Enzyme Deficiency (GBED). All these diseases are genetic, and are caused by single mutations in DNA that are easy to test for. The University of California-Davis performs the testing for the AQHA, using mane or tail hair or blood samples.
The panel has been available for years, but just recently AQHA began requiring that results of the panel be on file for stallions before their foals could be registered. This applied at first only to stallions that were bred to more than 25 mares, but after January 1, 2015, ALL breeding stallions will need to have the 5 panel test results on file. This requirement doesn’t restrict the use of stallions that may test positive for one or more of the diseases, but it does give mare owners valuable information to consider when making breeding decisions. Although the requirements for the panel testing only apply to breeding stallions, owners may request testing of any horse if they are interested or have concerns.
HYPP
HYPP stands for Hyperkalemic Periodic Paralysis. This disease affects the electrical impulses within the body that control muscle contraction. The defective gene results in clinical signs of muscle tremors and fasciculations. In some severe cases, horses may be unable to stand, or even unable to breathe. Horses can show symptoms with only one copy of the defective gene, but symptoms are often more severe if they have two copies of the mutation. This disease affects mostly halter horses, and can be traced back to the prolific stallion ‘Impressive’. Since Impressive lines were also used in Paint and Appaloosa halter breeding programs, HYPP is found in those breeds as well. AQHA does not allow registration of foals that test positive for two copies of the defective gene (H/H), but will allow registration of foals that are H/N: one defective and one normal gene.
PSSM 1
PSSM stands for Polysaccharide Storage Myopathy. This disease causes changes in the way sugars are stored and used by the muscles. It causes frequent episodes of ‘tying up’ if not properly controlled by a special diet and regular low intensity exercise. There are two types of PSSM. Type 1 is caused by a genetically identified mutation, which is testable. Type 2 is suspected to be genetic, but that mutation has not yet been identified by researchers. Most Quarter Horses with PSSM have type 1. Horses will show symptoms of PSSM type 1 with one or two copies of the mutation. Like HYPP, PSSM type 1 is more common in halter QHs than in other lines. Some QHs have been shown to have mutations for both HYPP and PSSM.
HERDA
HERDA stands for Hereditary Epidermal Regional Dermal Asthenia. Horses with HERDA have defective collagen, an important protein that is part of skin, cartilage, muscles, and tendons. The major clinical sign is skin that is easily injured, torn, or even sloughed off. The skin is also very slow to heal. There is no treatment for the condition, and horses that have it are often euthanized. Horses will only show symptoms if they have two copies of the mutation for HERDA. Horses with only one copy of the mutation are clinically normal. These animals are called ‘carriers’. They can pass copies of the mutation to their foals, and if one carrier is bred to another carrier, the foal might inherit the mutation from both parents and be symptomatic. HERDA is limited mostly to horses with reining and cutting horse bloodlines.
GBED
GBED stands for Glycogen Branching Enzyme Deficiency. Like PSSM, this disease also affects how sugars are stored, but in a different and more severe way. It results in abortions, stillborn foals, and foals that are alive but weak at birth and die or are euthanized soon after. Like with HERDA, horses may be carriers for GBED – if a horse has only one copy of the mutation it will be clinically normal. Paints and Appaloosas can also carry the GBED mutation.
MH
MH stands for Malignant Hyperthermia. This disease changes the way muscle cells handle calcium, and thus the metabolism of the cell. Horses with MH will appear normal most of the time, but have specific occasions when they show symptoms. During an attack, horses will have a very high fever, profuse sweating, high and irregular heart rate, high blood pressure, and rigid muscles. Attacks are triggered by certain anesthetic agents or stress, and are sometimes fatal. MH is believed to be less common than either HYPP or PSSM, but the percentage of affected horses is not yet known. Several breeds including Quarter Horses and Paints can be affected. Horses may be positive for both PSSM and MH together, and these animals appear to suffer from more severe episodes of tying up than horses that have PSSM alone.
Dec 23, 2014 | Breeding, Uncategorized
White Horse
Is there really such a thing as a white horse? Many gray horses start out nicely dappled, but then fade to nearly white as they age, and are incorrectly called ‘white’ by non-horsey folks. However there are a handful of other colors, each genetically different, that can result in a horse that looks white.
Blue-Eyed Creams Blue-eyed creams, (cremellos and perlinos), are horses that inherit two copies of the ‘cream’ gene. This gene is a color dilution factor, and when a horse inherits two copies, the result is a horse with blue eyes and an off white or cream colored coat. In the summer when they are slick and shiny these horses can appear white. Cremellos and perlinos can be difficult to tell apart without a genetic test to detect the differences in their underlying coat color. Cremellos are genetically chestnuts with two cream genes, and perlinos are genetically bay with two cream genes.
Lethal White Lethal white is an inherited condition usually seen in paint horse foals. These foals are born either all white, or mostly white, and like the name implies, they heartbreakingly will all die or have to be euthanized shortly after birth. The all white coat color in these foals is associated with a genetic mutation that also produces a problem in the nerves to the gastrointestinal tract, which interferes with motility and causes severe colic. There is no treatment for the condition. Usually, but not always, these foals are a product of breeding two overo paint horses. There is a genetic test available to screen potential breeding animals as carriers for the condition, and all responsible paint breeders should know the status of their breeding stock.
Sabino White Sabino is a description for a collection of white markings, and the word is used by multiple breeds. Horses labeled as sabinos often have extensive roaning, belly spots or large face markings and high white on the legs. Geneticists believe that there are probably several different genes that produce sabino type markings, but one mutation in particular has been identified, and named Sabino1. The Sabino1 gene is found mostly in Tennessee Walkers and Miniature horses, but also in some Paints, Shetland Ponies, and Pony of America’s. One copy of the Sabino1 gene produces the typical roaning, belly spots, high leg white, and large face markings. However, horses with two copies of Sabino1 are at least 90% white, and are referred to as ‘Sabino-white.’ These horses usually have pink skin and dark eyes. Sabino-white foals can look identical to Lethal-white foals, but they will be completely healthy, so it is important not to assume that all white foals should be euthanized at birth.
Dominant White Dominant white is caused by a variety of genetic mutations that affects pigment cells in skin and hair follicles. These mutations produce a horse with pink skin, white hair, and usually dark eyes. One particular such mutation, from the QH stallion GQ Santana, has been identified, and there is now a test available for it. Since it is a ‘dominant’ trait, only one copy of the mutation is needed to produce a white coat color. So far no horses with two copies of the mutation have been identified, so it is not known if foals/fetuses with two copies would be ‘viable’.
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